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NEW QUESTION: 1
All the following statements concerning a grantor-retained annuity trust (GRAT) or a grantor-retained unitrust (GRUT) are correct EXCEPT:
A. Post-transfer appreciation of property transferred to these trusts is excluded from the grantor's gross estate.
B. A grantor receives an annual payout from a GRUT that is based on the value that the trust grows or contracts to each year.
C. After the initial transfer to a GRAT, the grantor is prohibited from making additional contributions to the trust.
D. For gift tax purposes a grantor's retained interest in these trusts is valued at zero.
Answer: D

NEW QUESTION: 2
Select the class of Anti-diabetic medication that works in the specified organ to prevent hyperglycemia. Select all that applies. Muscle (F)
A. DPP4 Inhibitors
B. Sulfonylureas
C. Glucagon-like peptide-1 receptor agonists
D. Alpha- Glucosidase Inhibitors
E. Biguanide
F. Thiazolidinediones
G. SGLT2 inhibitors
Answer: F
Explanation:
Explanation
Sulfonylureas work in beta cells in the pancreas that are still functioning to enhance insulin secretion. Alpha- Glucosidase Inhibitors stop -glucosidase enzymes in the small intestine and delay digestion and absorption of starch and disaccharides which lowers the levels of glucose after meals. DPP4 blocks the degradation ofGLP-1, GIP, and a variety of other peptides, including brain natriuretic peptide. Glucagon-like peptide-1 receptor agonists work in various organs of the body. Glucagon-like peptide-1 receptor agonists enhance glucose homeostasis through: (i) stimulation of insulin secretion; (ii) inhibition of glucagon secretion; (iii) direct and indirect suppression of endogenous glucose production; (iv) suppression of appetite; (v) enhanced insulin sensitivity secondary to weight loss; (vi) delayed gastric emptying, resulting in decreased postprandial hyperglycaemia. Thiazolidinediones are the only true insulin-sensitising agents, exerting their effects in skeletal and cardiac muscle, liver, and adipose tissue. It ameliorates insulin resistance, decreases visceral fat.
Biguanides work in liver, muscle, adipose tissue via activation of AMP-activated protein kinase (AMPK) reduce hepatic glucose production. SGLT2 inhibitors work in the kidneys to inhibit sodium-glucose transport proteins to reabsorb glucose into the blood from muscle cells; overall this helps to improve insulin release from the beta cells of the pancreas.